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The reason for this isn't known but clearly exhibits that astrocytes do not oxidatively metabolize all the glucose they take up. The putative role for [Healthy Flow Blood formula](https://git.iweekyi.cn/karissawalters/healthy-flow-blood-health9039/wiki/The-Incredible-Science-behind-Eliud-Kipchoge%27s-1%3AFifty-Nine-Marathon) this lactate is open to debate. It could also be launched from the astrocyte as a form of vitality substrate that's immediately accessible to axons on the onset of elevated exercise. This is supported by knowledge from in vivo recordings of cat cortex wherein lactate sensors inserted into the cortex of a dwell cat briefly falls on the onset of experimentally induced activity. The non permanent dip is adopted by a rise in lactate in response to the steady stimulus (Hu and Wilson, 1997). The preliminary dip is assumed to be because of the onset of activity requiring an immediate enhance in delivery of substrate to the neurones. The lactate in the interstitial space meets this criterion. The latter enhance in lactate is presumably because of the mechanism whereby neurones signal to astrocytes of their increased power requirements, and the astrocytes respond by releasing lactate.
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